View source for Vascular permeability

'''Vascular permeability''', often in the form of '''capillary permeability''', characterizes the capacity of a blood vessel wall to allow the for the flow of small molecules (ions, water, nutrients) or even whole cells ([[lymphocyte]]s on their way to the site of [[inflammation]]) in and out of the vessel. [[Blood vessel]] walls are lined by a single layer of [[endothelial]] cells. The gaps between endothelial cells ([[cell junction]]s) are strictly regulated depending on the type and physiological state of the tissue.

An example of increased vascular permeability is in the [[Periodontal_disease#Initial_lesion|initial lesion of periodontal disease]], in which the [[plexus#Circulatory_system|gingival plexus]] becomes engorged and [[Vasodilation|dilated]], allowing large numbers of [[neutrophil]]s to [[leukocyte extravasation|extravasate]] and appear within the [[junctional epithelium]] and underlying [[connective tissue]].<ref name="P&S">Page, RC; Schroeder, HE. "Pathogenesis of Inflammatory Periodontal Disease: A Summary of Current Work." ''Lab Invest'' 1976;34(3):235-249</ref>

==References==
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==External links==
* {{MeshName|Vascular+Permeability}}

[[Category:Cardiovascular system]]

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