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...or|5-HT₃ receptor]]s, and slightly potentiates [[GABAA receptor|GABA_A]] and [[glycine receptor]]s.<ref name="pmid11020766">{{cite jou ...</ref> Indeed, in humans given 30% N₂O, benzodiazepine receptor antagonists reduced the subjective reports of feeling "high", but did not alter psycho-

| group1 = [[GABAA receptor|GABA_A receptor]] ...sub>2A</sub>]] & [[alpha-1 adrenergic receptor|α₁-adrenergic]] antagonists

| group1 = [[GABAA_receptor|GABA_A]] [[Positive_allosteric_modulator|PAM]]s | group7 = [[Melanin-concentrating hormone receptor 1|MCH₁ antagonists]]

| group7 = [[GABA A receptor|GABA_A]] [[GABRA5|α₅]] [[inverse agonist]]s

...= [[Dissociative]]s<br/>''<small>[[NMDA_receptor|NMDAR]] [[NMDA_antagonist|antagonists]]</small>'' ...br/>''<small>[[Muscarinic_acetylcholine_receptor|mAChR]] [[Anticholinergic|antagonists]]</small>''

...or|mGluR]]'' ([[Acamprosate]]){{·}} ''[[opioid antagonist|Opioid receptor antagonists]]'' ([[Naltrexone]], [[Nalmefene]]){{·}} [[Topiramate]]{{·}} ''[[Adrenerg ...I]]'' ([[Vanoxerine]]){{·}} [[Nocaine]]{{·}} ''[[gamma-Aminobutyric acid|GABA analogue]]'' ([[Vigabatrin]])

...controlled. For instance, when the release of the inhibitory transmitter GABA is reduced, the net effect is an increase in the excitability of the endoca * [[SR144528]], a CB₂ receptor antagonists

...[[Henri Laborit]] to use in studying the neurotransmitter [[GABA receptor|GABA]].<ref>{{cite journal | author = H. Laborit, J.M. Jouany, J. Gerald, F. Fab .../pjp/pdf/2004/1_43.pdf|format=PDF}}</ref> GHB is probably synthesized from GABA in GABAergic [[neurons]], and released when the neurons fire.<ref name="mec