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...s) which lack the enzyme to remove the phosphate once it has been added by kinases, and therefore attain high concentrations of the drug. Most of the kinase a

...es in AMPK activity and may be mediated by [[p38 mitogen-activated protein kinases|p38 MAPK]]- and [[Protein kinase C|PKC]]-dependent mechanisms."<ref>{{vcite

...ation of cytokines appear to occur via the [[p38 mitogen-activated protein kinases|p38 MAPK]]s (mitogen activated protein kinase) dependent pathway. Usually,

...talyze [[phosphorylation]] whereas [[phosphatase]]s dephosphorylate. These kinases and phosphatases regulate phosphate form and phosphate availability.

...y a suite ([[P38 mitogen-activated protein kinases|p38]]/p42/p44) of [[MAP kinases]] but also the [[PI3/PKB]] and [[MEK/ERK]] pathway (Galve-Roperh et al., 20

...s, [[protein phosphatase|phosphatase]]s, [[Phosphoinositide 3-kinase|lipid kinases]], and [[phospholipase C|hydrolases]].&nbsp; Some receptor-stimulated enzy ...|title=Mechanisms of regulation and function of G-protein-coupled receptor kinases|journal=World J Gastroenterol|volume=12|issue=48|pages=7753–7|year=2006|p

...-affinity receptor family for the neurotrophins, the Trk receptor tyrosine kinases such as [[TrkA]]. ==The Trk family of receptor tyrosine kinases==