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...lase]], which converts [[L-DOPA|<small>L</small>-DOPA]] into [[dopamine]]. Dopamine is a precursor for [[norepinephrine]] (noradrenaline) and subsequently [[ep * It is converted to [[α-methylnorepinephrine]] by [[dopamine beta-hydroxylase]] (DBH). α-methylnorepinephrine is an [[agonist]] of pres

|ImageFile2=Dopamine-3d-CPK.png ...cyclopedia.com/doc/1O87-ventraltegmentalarea.html Reference for VTA.</ref> Dopamine is also a [[neurohormone]] released by the [[hypothalamus]]. Its main funct

...on of renal arteries. Also, difficulty urinating is not uncommon, as alpha-agonists such as ephedrine constrict the internal urethral sphincter, mimicking the ...mode of therapeutic action involves norepinephrine and to a lesser degree dopamine, but it also releases some serotonin from presynaptic clefts. It should not

...5, 1985 | volume = 36| issue = 8| pages = 737–44| title = Effect of GABA agonists on the neurotoxicity and anticonvulsant activity of benzodiazepines| author ...}}</ref><ref name="Atack-2005"/> A single dose of diazepam modulates the [[dopamine]] system in similar ways to how morphine and [[ethanol|alcohol]] modulate t

...and [[norepinephrine]] receptors. It is its action on the [[D2 receptor|D2 dopamine]] and [[5HT1A|5-HT_1A receptor]]s that can cause some side effect {{Adrenergic agonists}}

...octopamine is by β-[[hydroxylation]] of [[tyramine]] via the [[enzyme]] [[dopamine β-hydroxylase]]. Under the trade names '''Epirenor''', '''Norden''', and ' [[Category:Alpha-adrenergic agonists]]

...e]], or as a [[norepinephrine releasing agent]] (NRA). It also acts as a [[dopamine releasing agent]] (DRA) to a lesser extent. [[Category:Alpha-adrenergic agonists]]

...no anticholinergic properties and is believed be devoid of any activity on dopamine, serotonin or histamin receptors. It is not addictive and tolerance does no ...eceptor antagonist), [[clonidine]] and other α₂-adrenoreceptor agonists, and [[mexiletine]] and other local anaesthetic analogues.

While there have been some papers that have linked concurrent stimulation of dopamine and CB1 receptors to an acute rise in cAMP production, it is accepted that ...ated changes to the membrane voltage have also been studied in detail. CB1 agonists reduce calcium influx by blocking the activity of voltage-dependent [[N-typ

...thways involving the μ₁ [[opioid receptor]], precipitating a [[dopamine]] release in the [[nucleus accumbens]]. The effects of the drug can be supp ...echanism for analgesic effects caused directly by THC or other cannabinoid agonists is not fully understood. Other effects include relaxation; [[euphoria]]; a

...e has been shown to act as an epsilon-opioid antagonist. Several selective agonists and antagonists are now available for the putative epsilon receptor,<ref na ...lesser degree than full agonists do. This slight release of serotonin and dopamine may contribute to the anti-depressant properties of buprenophine, especiall

...html NMDA receptor antagonists ketamine and PCP have direct effects on the dopamine D2 and serotonin 5-HT2receptorsimplications for models of schizophrenia](20 ...= August | pmid = 19391150 | doi = 10.1002/syn.20647}}</ref> as well as [[dopamine reuptake inhibitor]]s.{{Citation needed|date=April 2010}}

...of ''Salvia divinorum'' on mood. It is well-established that some k-opioid agonists can cause [[dysphoria]] in humans,<ref name="Rothman2000">[[#refRothman2000 ...''Salvia divinorum'', in line with the studied effects of other κ-opioid agonists,<ref name="Schenk2001">[[#refSchenk2001|Schenk 2001]], p.&nbsp;629–34.</r

[[Category:Dopamine reuptake inhibitors]] [[Category:Sigma agonists]]

...B receptor cause [[absence seizures]] in high doses, as do GHB and GABA(B) agonists.<ref>{{cite journal |author=Banerjee PK, Snead OC |title=Presynaptic gamma- ...oxone]] and [[naltrexone]]. Dynorphin may play a role in the inhibition of dopamine release via [[kappa opioid receptor]]s.<ref>{{cite journal |author=Mamelak