Eye movement desensitization and reprocessing

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Eye movement desensitization and reprocessing (EMDR) is a form of psychotherapy that was developed to resolve symptoms resulting from disturbing and unresolved life experiences. It uses a structured approach to address past, present, and future aspects of disturbing memories. The approach was developed by Francine Shapiro[1][2][3] to resolve the development of trauma-related disorders as resulting from exposure to a traumatic or distressing event, such as rape or military combat. Clinical trials have been conducted to assess EMDR's efficacy in the treatment of post-traumatic stress disorder (PTSD).[4][5][6][7] In some studies it has been shown to be equivalent to cognitive behavioral therapy and exposure therapies.[Unreliable medical source?][8][9][10][11] Although some clinicians may use EMDR for various problems, its research support is primarily for disorders stemming from distressing life experiences.[12][13]

Description of therapy

Although EMDR is established as an evidence-based treatment for PTSD[14][15][16][17][18][19][20][21][22] there are two main perspectives on EMDR therapy. First, Shapiro[1] proposed that although a number of different processes underlie EMDR, the eye movements add to the therapy's effectiveness by evoking neurological and physiological changes that may aid in the processing of the trauma memories being treated. The other perspective is that the eye movements are an epiphenomenon, unnecessary, and that EMDR is simply a form of desensitization.[23]

Theoretical basis

EMDR integrates elements of effective psychodynamic, imaginal exposure, cognitive therapy, interpersonal, experiential, physiological and somatic therapies. It also uses the unique element of bilateral stimulation (e.g. eye movements, tones, or tapping). According to Francine Shapiro's theory,[1][2] when a traumatic or distressing experience occurs, it may overwhelm usual ways of coping and the memory of the event is inadequately processed; the memory is dysfunctionally stored in an isolated memory network. When this memory network is activated, the individual may re-experience aspects of the original event, often resulting in inappropriate overreactions. This explains why people who have experienced or witnessed a traumatic incident may have recurring sensory flashbacks, thoughts, beliefs, or dreams. An unprocessed memory of a traumatic event can retain high levels of sensory and emotional intensity, even though many years may have passed.

EMDR uses a structured eight-phase approach and addresses the past, present, and future aspects of the dysfunctionally stored memory. During the processing phases of EMDR, the client attends to the disturbing memory in multiple brief sets of about 15–30 seconds, while simultaneously focusing on the dual attention stimulus (e.g., therapist-directed lateral eye movement, alternate hand-tapping, or bilateral auditory tones). Following each set of such dual attention, the client is asked what associative information was elicited during the procedure. This new material usually becomes the focus of the next set. This process of alternating dual attention and personal association is repeated many times during the session.

The theory is that EMDR works directly with memory networks and enhances information processing by forging associations between the distressing memory and more adaptive information contained in other semantic memory networks. It is thought that the distressing memory is transformed when new connections are forged with more positive and realistic information. This results in a transformation of the emotional, sensory, and cognitive components of the memory so that, when it is accessed, the individual is no longer distressed. Instead he/she recalls the incident with a new perspective, new insight, resolution of the cognitive distortions, elimination of emotional distress, and relief of related physiological arousal.[12]

When the distressing or traumatic event is an isolated, single incident, approximately three sessions are necessary for comprehensive treatment.[6][8] When multiple traumatic events contribute to a health problem—such as physical, sexual, or emotional abuse, parental neglect, severe illness, accident, injury, or health-related trauma that result in chronic impairment to health and well-being, or combat trauma, the time to heal may be longer,[24] and complex, multiple trauma may require many more sessions for the treatment to be complete and robust.[25]

Therapy process

The therapy process and procedures are according to Shapiro (2001)[2]

Phase I
In the first sessions, the patient's history and an overall treatment plan are discussed. During this process the therapist identifies and clarifies potential targets for EMDR. Target refers to a disturbing issue, event, feeling, or memory for use as an initial focus for EMDR. Maladaptive beliefs are also identified.
Phase II
Before beginning EMDR for the first time, it is recommended that the client identify a safe place, an image or memory that elicits comfortable feelings and a positive sense of self. This safe place can be used later to bring closure to an incomplete session or to help a client tolerate a particularly upsetting session.
Phase III
In developing a target for EMDR, prior to beginning the eye movement, a snapshot image is identified that represents the target and the disturbance associated with it. Using that image is a way to help the client focus on the target, a negative cognition (NC) is identified – a negative statement about the self that feels especially true when the client focuses on the target image. A positive cognition (PC) is also identified – a positive self-statement that is preferable to the negative cognition.
Phase IV
The therapist asks the patient to focus simultaneously on the image, the negative cognition, and the disturbing emotion or body sensation. Then the therapist usually asks the client to follow a moving object with his or her eyes; the object moves alternately from side to side so that the client's eyes also move back and forth. After a set of eye movements, the client is asked to report briefly on what has come up; this may be a thought, a feeling, a physical sensation, an image, a memory, or a change in any one of the above. In the initial instructions to the client, the therapist asks him or her to focus on this thought, and begins a new set of eye movements. Under certain conditions, however, the therapist directs the client to focus on the original target memory or on some other image, thought, feeling, fantasy, physical sensation, or memory. From time to time the therapist may query the client about her or his current level of distress. The desensitization phase ends when the SUDS (Subjective Units of Disturbance Scale) has reached 0 or 1.
Phase V
The "Installation Phase": the therapist asks the client about the positive cognition, if it's still valid. After Phase IV, the view of the client on the event/ the initial snapshot image may have changed dramatically. Another PC may be needed. Then the client is asked to "hold together" the snapshot and the (new) PC. Also the therapist asks, "How valid does the PC feel, on a scale from 1 to 7?" New sets of eye movement are issued.
Phase VI
The body scan: the therapist asks if anywhere in the client's body any pain, stress or discomfort is felt. If so, the client is asked to concentrate on the sore knee or whatever may arise and new sets are issued.
Phase VII
Debriefing: the therapist gives appropriate info and support.
Phase VIII
Re-evaluation: At the beginning of the next session, the client reviews the week, discussing any new sensations or experiences. The level of disturbance arising from the experiences targeted in the previous session is assessed. An objective of this phase is to ensure the processing of all relevant historical events.

EMDR also uses a three-pronged approach, to address past, present and future aspects of the targeted memory.

Vocabulary

The following terms are described in Shapiro's 2001 text:[2]

Information processing
During information processing, a physiologically-based system sorts new (perceptual) information, makes connections between new information and other information already stored in associated memory networks, encodes the material, and stores it in memory.
Adaptive resolution
When information processing is complete, learning takes place, and information is stored in memory with appropriate emotion. The new information is therefore available to guide future action.
Dysfunctionally Stored Information
When information processing is incomplete, the information is not connected to more adaptive information, and it is stored in a memory network with a high negative emotional charge. It can cause reactivity and can be the cause of various symptoms.
Reprocessing
During reprocessing in EMDR, new associative links are forged between dysfunctionally stored information and adaptive information, resulting in complete information processing and adaptive resolution.
Memory networks
Neurobiological associations of related memories, sensations, images, thoughts, and emotions.
Target memory
The memory of a distressing or traumatic event, which still causes current distress, and which has been selected to be targeted during EMDR treatment.
Memory components
All components of the target memory are accessed during phase three to ensure that the memory network is fully activated. These components include the image, cognitions, emotions, and body sensations.
Validity of cognition (VOC) scale
VOC ratings are used in EMDR to measure baseline validity of the positive cognition during phase three, and to assess progress being made, where 1 = not true, and 7 = completely true.
Subjective units of disturbance (SUD) scale
SUD ratings are used in EMDR, exposure therapies, and other treatments to measure baseline emotional or physical pain and also to assess progress being made. This is a personal measurement of distress, where 0 = no distress, and 10 = worst distress possible.
Interweave
A specific strategy used by the clinician to assist processing if the client appears to be having difficulty accessing more adaptive information. Ideally, the interweave contains needed information that would have been available except for blockage of inner pathways by trauma responses.

Mechanism

The theory underlying EMDR treatment is that it works by helping the sufferer process distressing memories more fully which reduces the distress. EMDR is based on a theoretical information processing model which posits that symptoms arise when events are inadequately processed, and can be eradicated when the memory is fully processed. It is an integrative therapy, synthesizing elements of many traditional psychological orientations, such as psychodynamic, cognitive behavioral, experiential, physiological, and interpersonal therapies.[26]

Overview

EMDR's most unique aspect is an unusual component of bilateral stimulation of the brain, such as eye movement, bilateral sound, or bilateral tactile stimulation coupled with cognitions, visualized images and body sensation. EMDR also utilizes dual attention awareness to allow the individual to vacillate between the traumatic material and the safety of the present moment. This prevents retraumatization from exposure to the disturbing memory. As EMDR is an integrative therapy which combines elements of cognitive behavioral and psychodynamic therapies to desensitize traumatic memories, some individuals have criticized EMDR and consider the use of eye movement to be an unnecessary component of treatment.[27][28] However, recent studies have examined the effects of eye movement and have found that eye movement in EMDR decreases the vividness and/or negative emotions associated with autobiographical memories,[29][30][31][32] enhance the retrieval of episodic memories,[33] increase cognitive flexibility,[34] and correlate with decreases in heart rate, skin conductance, and an increased finger temperature.[35][36] These physiological changes associated with EMDR are consistent with earlier research on physiological changes associated with EMDR.[37] Also recent studies that have removed the eye movement component from the method have found the procedure to be less effective.[38]

Hypotheses

There is no definitive explanation as to how EMDR works. There is some empirical support for three explanations regarding how an external stimulus such as eye movement can facilitate the processing of traumatic memories.

Failure to process episodic memory

The first hypothesis views PTSD as a failure by the individual to process episodic memory;[2][39] the bilateral eye movement involved in EMDR facilitate interaction between the brain's hemispheres, which then improves the processing of trauma-related memories. This hypothesis is supported by a study that tested the effects of eye movement on the ability to retrieve episodic memory. The study found better recall following a horizontal eye movement task compared to that following no eye movement or a vertical eye movement task.[33]

Similarity to REM sleep

A second hypothesis suggests that eye movement facilitates processing of trauma memories by activating a neurobiological state similar to REM sleep wherein associative links to episodic memories are formed and these memories are then integrated into general semantic networks. Stickgold proposed that PTSD occurs when an event is sufficiently arousing to prevent its transfer from encoding from an episodic memory to a semantic memory.[39] As a result of high arousal levels, associations between the traumatic event and other related events fail to develop. He argues that the attentional redirecting in EMDR induces a neurobiological state similar to REM sleep. He then reviews the research that suggests that REM sleep enhances processing of episodic memory through the preferential activation of weak associative and semantic links. Thus in EMDR trauma-related information that is closely associated with a target event is weakened and ancillary information loosely related to the event is strengthened, allowing the integration of trauma-related material with other loosely associated events in the person’s life. Support for this argument comes from a study that found that, compared to eye fixation, eye movement promoted attentional flexibility and increased preparedness to process metaphorical material.[34]

Orienting response

A third hypothesis links the eye movement in EMDR with the orienting response.[40] MacCulloch and Feldman argued that eye movement triggers the investigation component of the orienting response, which can either produce avoidance behaviour or inhibit avoidance responses. Inhibiting avoidance behaviour includes reducing both negative somatic responses and cognitive changes that would allow fresh investigatory behaviour to commence. MacCulloch and Feldman proposed that initially when danger is identified there is a negative affect response. However a second part of the orienting response is to scan for further danger, and this investigatory reflex seems to accompany a positive physical response. In the authors’ opinion, eye movement induces this investigatory reflex and produces a relaxation response. A relaxation response was, in fact, found in a study that investigated the autonomic responses of participants when they were engaged in an eye movement task as part of EMDR treatment[37] and when participants focused on negative memories while engaging in eye movement[citation needed]. However there is not a differential effect of eye movement on a relaxation response when participants focused on positive memories.[30] This supports the hypothesis that eye movement is an orienting response mechanism rather than a simple relaxation mechanism. In addition, recent research that has examined the physiological correlates of eye movement in EMDR has found that a clear orienting response pattern of psycho-physiological de-arousal occurs when eye movement begins, and this de-arousal is characteristic of the physiological changes that occur when an orienting response is elicited.[36]

Further data consistent with the orienting response hypothesis was the finding that EMDR treatment was associated with increased left pre-frontal hemisphere activation.[41][42] Investigatory and approach behavior has been shown to be associated with the anterior left hemisphere regions.[43]

Empirical evidence and comparison

Based on the evidence of randomised controlled research trials the most recent scientific review by Cochrane[15] rated EMDR in the highest category of effectiveness for the treatment of PTSD, and the International Society of Stress Studies practice guidelines[19] has recently ranked EMDR as an evidence-based level A treatment for PTSD in adults. This status is reflected in a number of international guidelines where EMDR is a recommended treatment for trauma.[16][17][18][19][20][21][22]

EMDR was found in the first ever meta-analysis of PTSD to be equally effective as exposure therapy and SSRIs.[44] Two recent meta-analyses concluded that traditional exposure therapy and EMDR have equivalent effects both immediately after treatment and at follow-up.[45][46] A 2007 meta-analysis looked at 38 randomized controlled trials for PTSD treatment and concluded that the first-line psychological treatment for PTSD should be Trauma-Focused CBT or EMDR.[47] The next year, a meta-analysis concluded that all "bona fide" treatments were equally effective, but this paper was soon dismissed for its arbitrary selection of which treatments were "bona fide".[48] An APA comparative review in CNS Spectrums found EMDR to be of similar efficacy to other exposure therapies, and superior to SSRI, problem-centred therapy or treatment as usual.[49]

Typical treatments

EMDR has been demonstrated to have significant advantages over usual treatment for PTSD in an HMO setting, and improvement was maintained at a six month follow-up.[50][51] EMDR has been shown to be effective on measures of trauma, depression and anxiety in women and men who have been sexually abused as children.[52][52][53]

Medication

To date EMDR has only been compared directly to medication in one study. Van der Kolk et al.[11] found EMDR to be more effective than the SSRI fluoxetine in treating trauma, especially six months post-treatment. The study also suggests a role for SSRIs as a reliable first-line intervention.

Exposure treatment

EMDR proved significantly better than stress inoculation training with prolonged exposure in a study with 24 participants diagnosed with post traumatic stress disorder[9] Although reduction in symptom severity was equivalent post treatment, at follow-up, EMDR lead to greater gains on both self report and observer rated measures of PTSD and self report measures of depression. In another study of 22 participants who had also been diagnosed with PTSD, both EMDR and prolonged exposure were found to be effective post treatment.[8] Participants receiving EMDR appeared to improve more quickly; 70% reached a level of clinically significant improvement in PTSD symptoms after three EMDR sessions compared to 17% in the prolonged exposure condition.

EMDR was also found to work more quickly than exposure based treatments in a larger trial with 105 participants.[10] At a fifteen-month follow-up, gains for both treatments were generally maintained. The only significant difference at follow-up was an improvement in depression according to an independent observer in favour of EMDR.

EMDR and Prolonged Exposure (PE) were found to be equivalently efficacious and both superior to a waitlist control in a controlled trial of 74 female rape survivors.[Unreliable medical source?][54] Measures used by blind assessors included PTSD, depression, dissociation and state anxiety. Unlike other studies noted above, there was no difference between the active treatments in rate of improvement. However EMDR seemed to do adequately well despite utilising no homework tasks and less exposure. The study met the highest criteria for methodological rigour proposed by Foa and Meadows.[55]

Cognitive behavioral therapy

Although most studies show EMDR and CBT to be equally effective in the treatment of PTSD, one study reported an opposite effect.[56] Analysis of changes in symptoms for the 15 participants who completed treatment indicated greater reductions on symptom measures of avoidance and re-experiencing for imaginal exposure treatment over EMDR but equivalent reductions on hyperarousal. However, there were no differences between the two treatments in the intent to treat analysis and there was no differences between the two treatments on percentage of people with PTSD diagnosis at follow-up.

The effectiveness of EMDR compared to cognitive behavioral therapy (CBT) is not limited to English speaking cultures. In a study involving Iranian girls who had been sexually abused, EMDR was found to be significantly more efficient than CBT, with similar treatment effects achieved within fewer sessions of EMDR than CBT.[57]

Other applications

Although controlled research has concentrated on the application of EMDR to PTSD, a number of studies have investigated EMDR’s efficacy with other anxiety disorders as well as numerous reports of diverse clinical applications.

Personality disorders

Case reports have been published on the application of EMDR to the treatment of (a) personality disorders (Fensterheim, 1996a; Korn & Leeds, in press; Manfield, 1998), (b) dissociative disorders (Fine & Berkowitz, 2001; Lazrove & Fine, 1996; Paulsen, 1995; Twombly, 2000), (c) a variety of anxiety disorders[58] (De Jongh & Ten Broeke, 1998; Goldstein & Feske, 1994; Lovett, 199; Nadler, 1996; Shapiro & Forrest, 1997) and (d) somatoform disorders (Brown, Mcgoldrick, & Buchanan, 1997; Grant & Threlfo, 2002). However, controlled research is needed to evaluate the efficacy of these applications.

Body dysmorphic disorder

In designing the research the entire EMDR protocol should be evaluated within the context of the potential special needs of the particular population. For instance, Brown et al. (1997) evaluated the application of EMDR in seven consecutive cases of Body Dysmorphic Disorder (BDD), which has been reported to necessitate 8 to 20 sessions of cognitive behavior therapy with varying success rates (Neziroglu, McKay, Todaro, & Yaryura-Tobias, 1996; Beale et al., 1996; Wilhelm, Otto, Lohr, & Deckersbach, 1999). In contrast, Brown et al. reported the elimination of BDD in five of the seven consecutive cases in one to three sessions of EMDR through the processing of the etiological memory. While this result indicates the EMDR holds promise for the treatment of this disorder, future controlled research should include a greater number of sessions in order to evaluate the more comprehensive clinical picture.

Depression

EMDR can work on a multitude of problems that are less complex than PTSD. One of these is uncomplicated depression. The EMDR Casebook by Philip Manfield, has documented several case studies in which EMDR was used. In the case about uncomplicated depression, Manfield was able to help his client, George, resolve several childhood issues that have plagued his adult life. Moreover, EMDR can work for postpartum depression. By having the client target a distinctive memory and work through it with a series of eye movements, the client is then able to achieve a positive cognition.[59]

In children

EMDR has been used on children to treat a variety of conditions.[60][Unreliable medical source?][61][Unreliable medical source?] It has been used in the treatment of children who have experienced trauma and complex trauma.[60][61]

It is often cited as a component in the treatment of complex post-traumatic stress disorder,[62] emotional dysregulation, and in the treatment of children exposed to chronic early maltreatment that is related to attachment disorder. It is recognised by the UK National Institute for Health & Clinical Excellence (NICE) Guidelines as a treatment for PTSD.[63]

For personal improvement

EMDR has also been used in performance and creativity enhancement with athletes and stage performers.[64][Unreliable medical source?]

Controversy over mechanisms and effectiveness

The working mechanisms that underlie the effectiveness of EMDR, and whether the eye movement component in EMDR contributes to its clinical effectiveness are still points of uncertainty and contentious debate.[citation needed]

EMDR has generated a great deal of controversy since its inception in 1989. Critics of EMDR argue that the eye movements do not play a central role, that the mechanisms of eye movements are speculative, and that the theory leading to the practice is not falsifiable and therefore not amenable to scientific enquiry.[27][65]

Although one meta-analysis concluded that EMDR is not as effective, or as long lasting, as traditional exposure therapy,[66] several other researchers using meta-analysis have found EMDR to be at least equivalent in effect size to specific exposure therapies.[44][45][47][46]

Exposure

Several papers have highlighted key differences between EMDR and traditional exposure treatments.[67][68] However, despite the treatment procedures being quite different between EMDR and traditional exposure therapy, some authors[16][69] continue to argue that the main effective component in EMDR is exposure. Other authors argue that EMDR is a complex therapy with many elements. Thus, it is too simple to assume that any one mechanism of action is responsible for positive treatment effects.[70]

Although exposure to traumatic memories occurs in EMDR, the form of exposure is different as it is short and intermittent as opposed to uninterrupted and prolonged, it is non-directive and allows for free association, and reliving is not a requirement of effective therapy like it is in traditional exposure.[71] Recent research by Lee and colleagues[38][72] has found key differences in the crucial processes of EMDR and traditional exposure. They have demonstrated that reliving within EMDR is not associated with symptom improvement. Rather greater improvement in trauma symptoms occur when trauma material is processed in a more detached or distant way. The distraction created by the eye movements in EMDR also distinguishes EMDR processes from those in traditional exposure. Although it has been shown recently that some level of distraction in exposure assists in anxiety reduction,[73] traditionally distraction tasks in exposure were thought to interfere with treatment effectiveness.[74][75]

In sum, the exposure that occurs in EMDR should, according to the assumptions of emotional processing theory,[76] sensitise rather than desensitise and decrease the fear and distress associated with traumatic memories. However, EMDR is effective, therefore, processes other than imaginal exposure must play a role in the effectiveness of EMDR in the treatment of PTSD.

Eye movements

An early critical review and metanalysis that looked at the contribution of eye movement to treatment effectiveness in EMDR concluded that eye movement is not necessary to the treatment effect.[23][77] However, recent research has demonstrated that when the eye movement component of EMDR is removed from the method the procedure is less effective.[38] This finding supports previous research that has demonstrated that EMDR with eye movement is more effective than treatment conditions that do not use eye movement in the method,[78][79] or instead use a dual attention task such as tapping.[37]

MacCulloch (2006) argued that the eye movement makes a unique contribution to EMDR,[80] whereas Salkovskis (2002) reported that the eye movement is irrelevant and that the effectiveness of the procedure is solely due to it sharing similar properties to cognitive behavioral therapies, such as desensitization and exposure.[81]

Effect of eye movement on memory, cognitive processes, and physiology

Although the necessity of eye movement in EMDR is still a point of controversy and contentious debate, a separate body of research has examined the effects of eye movement on physiology, memory, and cognition during the process of EMDR. To date the research in this area has demonstrated that eye movement decreases the vividness and/or emotional valence of autobiographical memories,[29][31][32] they enhance the retrieval of episodic memories,[33] produce a physiological relaxation effect, similar to that which is characteristically seen when an orienting response is elicited,[30][36][82] and they have been found to increase cognitive flexibility.[34] Although a wide range of researchers have proposed various models and theories to explain the effect of eye movement, and the possible role that eye movement may play in the process of EMDR, to date, no single model or theory exists that can explain all of the above mentioned findings. Further research is therefore required in this area.

References

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  1. ↑ 1.0 1.1 1.2 Shapiro F (1995). Eye movement desensitization and reprocessing: basic principles, protocols, and procedures. New York: Guilford Press. p. 398. ISBN 0-89862-960-8. 
  2. ↑ 2.0 2.1 2.2 2.3 2.4 Shapiro F (2001). EMDR: Eye Movement Desensitization of Reprocessing: Basic Principles, Protocols and Procedures (2nd ed.). New York: Guilford Press. p. 472. ISBN 1-57230-672-6. OCLC 46678584. 
  3. ↑ Shapiro, Francine (2002). EMDR as an Integrative Psychotherapy Approach: Experts of Diverse Orientations Explore the Paradigm Prism. Washington, DC: American Psychological Association. ISBN 1-55798-922-2. OCLC 48958394. 
  4. ↑ Högberg, G.; Pagani, M.; Sundin, O.; Soares, J.; Aberg-Wistedt, A.; Tärnell, B.; Hällström, T. (2007). "On treatment with eye movement desensitization and reprocessing of chronic post-traumatic stress disorder in public transportation workers--a randomized controlled trial". Nordic journal of psychiatry. 61 (1): 54–61. doi:10.1080/08039480601129408. PMID 17365790.  edit
  5. ↑ Rothbaum, BO (1997). "A controlled study of eye movement desensitization and reprocessing in the treatment of posttraumatic stress disordered sexual assault victims". Bulletin of the Menninger Clinic. 61 (3): 317–34. PMID 9260344.  edit
  6. ↑ 6.0 6.1 Wilson, SA; Becker, LA; Tinker, RH (1995). "Eye movement desensitization and reprocessing (EMDR) treatment for psychologically traumatized individuals". Journal of consulting and clinical psychology. 63 (6): 928–37. doi:10.1037/0022-006X.63.6.928. PMID 8543715.  edit
  7. ↑ Wilson, SA; Becker, LA; Tinker, RH (1997). "Fifteen-month follow-up of eye movement desensitization and reprocessing (EMDR) treatment for posttraumatic stress disorder and psychological trauma". Journal of consulting and clinical psychology. 65 (6): 1047–56. doi:10.1037/0022-006X.65.6.1047. PMID 9420367.  edit
  8. ↑ 8.0 8.1 8.2 Ironson, G.; Freund, B.; Strauss, J.; Williams, J. (2002). "Comparison of two treatments for traumatic stress: a community-based study of EMDR and prolonged exposure". Journal of clinical psychology. 58 (1): 113–128. doi:10.1002/jclp.1132. PMID 11748600.  edit
  9. ↑ 9.0 9.1 Lee, C.; Gavriel, H.; Drummond, P.; Richards, J.; Greenwald, R. (2002). "Treatment of PTSD: stress inoculation training with prolonged exposure compared to EMDR". Journal of clinical psychology. 58 (9): 1071–1089. doi:10.1002/jclp.10039. PMID 12209866.  edit
  10. ↑ 10.0 10.1 Power, K.; McGoldrick, T.; Brown, K.; Buchanan, R.; Sharp, D.; Swanson, V.; Karatzias, A. (2002). "A controlled comparison of eye movement desensitization and reprocessing versus exposure plus cognitive restructuring versus waiting list in the treatment of post-traumatic stress disorder". Clinical Psychology & Psychotherapy. 9: 299. doi:10.1002/cpp.341.  edit
  11. ↑ 11.0 11.1 Van Der Kolk, BA; Spinazzola, J; Blaustein, ME; Hopper, JW; Hopper, EK; Korn, DL; Simpson, WB (2007). "A randomized clinical trial of eye movement desensitization and reprocessing (EMDR), fluoxetine, and pill placebo in the treatment of posttraumatic stress disorder: treatment effects and long-term maintenance". The Journal of clinical psychiatry. 68 (1): 37–46. doi:10.4088/JCP.v68n0105. PMID 17284128.  edit
  12. ↑ 12.0 12.1 Maxfield, L. (2003). "Clinical Implications and Recommendations Arising from EMDR Research Findings". Journal of Trauma Practice. 2: 61–81. doi:10.1300/J189v02n01_04.  edit
  13. ↑ Maxfield L; Shapiro F; Kaslow FW (2007). Handbook of EMDR and Family Therapy Processes. New York: Wiley. p. 504. ISBN 0471709476. 
  14. ↑ "Practice Guideline for the Treatment of Patients with Acute Stress Disorder and Posttraumatic Stress Disorder". 1. 2006. doi:10.1176/appi.books.9780890423363.52257.  edit
  15. ↑ 15.0 15.1 Bisson, J.; Andrew, M.; Bisson, J. (2007). "Psychological treatment of post-traumatic stress disorder (PTSD)". Cochrane database of systematic reviews (Online) (3): CD003388. doi:10.1002/14651858.CD003388.pub3. PMID 17636720.  edit
  16. ↑ 16.0 16.1 16.2 Australian Centre for Posttraumatic Mental Health. (2007). Australian guidelines for the treatment of adults with acute stress disorder and post traumatic stress disorder. Melbourne, Victoria: ACPTMH. ISBN 978-0-9752246-6-3. 
  17. ↑ 17.0 17.1 National Institute for Clinical Excellence (2005). "Post traumatic stress disorder (PTSD): The management of adults and children in primary and secondary care". London: NICE Guidelines. 
  18. ↑ 18.0 18.1 Dutch National Steering Committee Guidelines Mental Health and Care (2003). "Guidelines for the diagnosis treatment and management of adult clients with an anxiety disorder". Utrecht, Netherlands: The Dutch Institute for Healthcare Improvement (CBO). 
  19. ↑ 19.0 19.1 19.2 Foa EB; Keane TM; Friedman MJ (2009). "Effective treatments for PTST: Practice guidelines of the International Society for Traumatic Stress Studies". New York: Guilford Press. 
  20. ↑ 20.0 20.1 Foa EB; Keane TM; Friedman MJ (2000). "Effective treatments for PTST: Practice guidelines of the International Society for Traumatic Stress Studies". New York: Guilford Press. 
  21. ↑ 21.0 21.1 Bleich A; Kolter M; Kutz E; Shaley A (2002). "A position paper of the (Israeli) National Council for Mental Health: Guidelines for the assessment and professional intervention with terror victims in the hospital and the community". Jerusalem. 
  22. ↑ 22.0 22.1 United Kingdom Department of Health (2001). "Treatment choice in psychological therapies and counseling evidence based on clinical practice guideline". London: Author. 
  23. ↑ 23.0 23.1 Davidson, PR; Parker, KC (2001). "Eye movement desensitization and reprocessing (EMDR): a meta-analysis". Journal of consulting and clinical psychology. 69 (2): 305–16. doi:10.1037/0022-006X.69.2.305. PMID 11393607.  edit
  24. ↑ Phillips M (2000). Finding the Energy to Heal: How EMDR, hypnosis, TFT, imagery, and body focused therapy can help restore the mind body health. New York: W.W. Norton. 
  25. ↑ Carlson, J.; Chemtob, C.; Rusnak, K.; Hedlund, N.; Muraoka, M. (1998). "Eye movement desensitization and reprocessing (EDMR) treatment for combat-related posttraumatic stress disorder". Journal of traumatic stress. 11 (1): 3–24. doi:10.1023/A:1024448814268. PMID 9479673.  edit
  26. ↑ Shapiro, F.; Maxfield, L. (2002). "Eye Movement Desensitization and Reprocessing (EMDR): information processing in the treatment of trauma". Journal of clinical psychology. 58 (8): 933–946. doi:10.1002/jclp.10068. PMID 12115716.  edit
  27. ↑ 27.0 27.1 Herbert, JD; Lilienfeld, SO; Lohr, JM; Montgomery, RW; O'Donohue, WT; Rosen, GM; Tolin, DF (2000). "Science and pseudoscience in the development of eye movement desensitization and reprocessing: implications for clinical psychology". Clinical psychology review. 20 (8): 945–71. doi:10.1016/S0272-7358(99)00017-3. PMID 11098395.  edit
  28. ↑ Sikes, C.; Sikes, V. (2003). "EMDR: Why the Controversy?". Traumatology. 9: 169. doi:10.1177/153476560300900304.  edit
  29. ↑ 29.0 29.1 Andrade, J; Kavanagh, D; Baddeley, A (1997). "Eye-movements and visual imagery: a working memory approach to the treatment of post-traumatic stress disorder". The British journal of clinical psychology / the British Psychological Society. 36 ( Pt 2): 209–23. PMID 9167862.  edit
  30. ↑ 30.0 30.1 30.2 Barrowcliff, A.; Gray, N.; Freeman, T.; MacCulloch, M. (2004). "Eye-movements reduce the vividness, emotional valence and electrodermal arousal associated with negative autobiographical memories". Journal of Forensic Psychiatry and Psychology. 15: 325. doi:10.1080/14789940410001673042.  edit
  31. ↑ 31.0 31.1 Kavanagh, D. J.; Freese, S.; Andrade, J.; May, J. (2001). "Effects of visuospatial tasks on desensitization to emotive memories". British Journal of Clinical Psychology. 40 (Pt 3): 267. doi:10.1348/014466501163689. PMID 11593955.  edit
  32. ↑ 32.0 32.1 Van Den Hout, M; Muris, P; Salemink, E; Kindt, M (2001). "Autobiographical memories become less vivid and emotional after eye movements". The British journal of clinical psychology / the British Psychological Society. 40 (Pt 2): 121–30. doi:10.1348/014466501163571. PMID 11446234.  edit
  33. ↑ 33.0 33.1 33.2 Christman, SD; Garvey, KJ; Propper, RE; Phaneuf, KA (2003). "Bilateral eye movements enhance the retrieval of episodic memories". Neuropsychology. 17 (2): 221–9. doi:10.1037/0894-4105.17.2.221. PMID 12803427.  edit
  34. ↑ 34.0 34.1 34.2 Kuiken, D.; Bears, M.; Miall, D.; Smith, L. (2001). "Eye Movement Desensitization Reprocessing Facilitates Attentional Orienting". Imagination, Cognition and Personality. 21: 3. doi:10.2190/L8JX-PGLC-B72R-KD7X.  edit
  35. ↑ Elofsson, U.; Von Schèele, B.; Theorell, T.; Söndergaard, H. (2008). "Physiological correlates of eye movement desensitization and reprocessing". Journal of anxiety disorders. 22 (4): 622–634. doi:10.1016/j.janxdis.2007.05.012. PMID 17604948.  edit
  36. ↑ 36.0 36.1 36.2 Sack, M.; Lempa, W.; Steinmetz, A.; Lamprecht, F.; Hofmann, A. (2008). "Alterations in autonomic tone during trauma exposure using eye movement desensitization and reprocessing (EMDR)--results of a preliminary investigation". Journal of anxiety disorders. 22 (7): 1264–1271. doi:10.1016/j.janxdis.2008.01.007. PMID 18314305.  edit
  37. ↑ 37.0 37.1 37.2 Wilson, DL; Silver, SM; Covi, WG; Foster, S (1996). "Eye movement desensitization and reprocessing: effectiveness and autonomic correlates". Journal of behavior therapy and experimental psychiatry. 27 (3): 219–29. doi:10.1016/S0005-7916(96)00026-2. PMID 8959423.  edit
  38. ↑ 38.0 38.1 38.2 Lee, C.; Drummond, P. (2008). "Effects of eye movement versus therapist instructions on the processing of distressing memories". Journal of anxiety disorders. 22 (5): 801–808. doi:10.1016/j.janxdis.2007.08.007. PMID 17890048.  edit
  39. ↑ 39.0 39.1 Stickgold, R. (2002). "EMDR: a putative neurobiological mechanism of action". Journal of clinical psychology. 58 (1): 61–75. doi:10.1002/jclp.1129. PMID 11748597.  edit
  40. ↑ MacCulloch, M. J.; Feldman, P. (1996). "Eye movement desensitisation treatment utilises the positive visceral element of the investigatory reflex to inhibit the memories of post- traumatic stress disorder: a theoretical analysis". The British Journal of Psychiatry. 169 (5): 571. doi:10.1192/bjp.169.5.571. PMID 8932885.  edit
  41. ↑ Lansing, K.; Amen, D.; Hanks, C.; Rudy, L. (2005). "High-resolution brain SPECT imaging and eye movement desensitization and reprocessing in police officers with PTSD". The Journal of neuropsychiatry and clinical neurosciences. 17 (4): 526–532. doi:10.1176/appi.neuropsych.17.4.526. PMID 16387993.  edit
  42. ↑ Levin, P; Lazrove, S; Van Der Kolk, B (1999). "What psychological testing and neuroimaging tell us about the treatment of Posttraumatic Stress Disorder by Eye Movement Desensitization and Reprocessing". Journal of anxiety disorders. 13 (1-2): 159–72. doi:10.1016/S0887-6185(98)00045-0. PMID 10225506.  edit
  43. ↑ Kinsbourne M (1978). "Evolution of language in relation to lateral action". In M. Kinsbourne. Asymmetrical function of the brain. New York: Cambridge University Press. pp. 553–556.  Missing or empty |title= (help)
  44. ↑ 44.0 44.1 doi:10.1002/(SICI)1099-0879(199809)5:3<126::AID-CPP153>3.0.CO;2-H
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  45. ↑ 45.0 45.1 Bradley, R.; Greene, J.; Russ, E.; Dutra, L.; Westen, D. (2005). "A multidimensional meta-analysis of psychotherapy for PTSD". The American journal of psychiatry. 162 (2): 214–227. doi:10.1176/appi.ajp.162.2.214. PMID 15677582.  edit
  46. ↑ 46.0 46.1 Seidler, G.; Wagner, F. (2006). "Comparing the efficacy of EMDR and trauma-focused cognitive-behavioral therapy in the treatment of PTSD: a meta-analytic study". Psychological medicine. 36 (11): 1515–1522. doi:10.1017/S0033291706007963. PMID 16740177.  edit
  47. ↑ 47.0 47.1 Bisson, J. I.; Ehlers, A.; Matthews, R.; Pilling, S.; Richards, D.; Turner, S. (2007). "Psychological treatments for chronic post-traumatic stress disorder: Systematic review and meta-analysis". The British Journal of Psychiatry. 190: 97. doi:10.1192/bjp.bp.106.021402. PMID 17267924.  edit
  48. ↑ Ehlers, A.; Bisson, J.; Clark, D.; Creamer, M.; Pilling, S.; Richards, D.; Schnurr, P.; Turner, S.; Yule, W. (2010). "Do all psychological treatments really work the same in posttraumatic stress disorder?". Clinical psychology review. 30 (2): 269–276. doi:10.1016/j.cpr.2009.12.001. PMC 2852651 Freely accessible. PMID 20051310.  edit
  49. ↑ Cloitre, M (2009). "Effective psychotherapies for posttraumatic stress disorder: a review and critique". CNS spectrums. 14 (1 Suppl 1): 32–43. PMID 19169192.  edit
  50. ↑ Marcus S (1997). "Controlled study of treatment of PTSD using EMDR in an HMO setting". Psychotherapy. 34: 307–315. 
  51. ↑ Marcus, S.; Marquis, P.; Sakai, C. (2004). "Three- and 6-Month Follow-Up of EMDR Treatment of PTSD in an HMO Setting". International Journal of Stress Management. 11: 195. doi:10.1037/1072-5245.11.3.195.  edit
  52. ↑ 52.0 52.1 Edmond T; Rubin A; Wambacj K (1999). "The effectiveness of EMDR with adult female survivors of childhood sexual abuse" (pdf). Social Work Research. 23 (2): 103–116. 
  53. ↑ Edmond, T.; Sloan, L.; McCarty, D. (2004). "Sexual Abuse Survivors' Perceptions of the Effectiveness of EMDR and Eclectic Therapy". Research on Social Work Practice. 14: 259. doi:10.1177/1049731504265830.  edit
  54. ↑ Rothbaum, B.; Astin, M.; Marsteller, F. (2005). "Prolonged Exposure versus Eye Movement Desensitization and Reprocessing (EMDR) for PTSD rape victims". Journal of traumatic stress. 18 (6): 607–616. doi:10.1002/jts.20069. PMID 16382428.  edit
  55. ↑ Foa, E.; Meadows, E. (1997). "Psychosocial treatments for posttraumatic stress disorder: a critical review". Annual review of psychology. 48: 449–480. doi:10.1146/annurev.psych.48.1.449. PMID 9046566.  edit
  56. ↑ Taylor, S; Thordarson, DS; Maxfield, L; Fedoroff, IC; Lovell, K; Ogrodniczuk, J (2003). "Comparative efficacy, speed, and adverse effects of three PTSD treatments: exposure therapy, EMDR, and relaxation training". Journal of consulting and clinical psychology. 71 (2): 330–8. doi:10.1037/0022-006X.71.2.330. PMID 12699027.  edit
  57. ↑ Jaberghaderi, N.; Greenwald, R.; Rubin, A.; Zand, S. O.; Dolatabadi, S. (2004). "A comparison of CBT and EMDR for sexually-abused Iranian girls". Clinical Psychology & Psychotherapy. 11: 358. doi:10.1002/cpp.395.  edit
  58. ↑ De Jongh, A; Ten Broeke, E; Renssen, MR (1999). "Treatment of specific phobias with Eye Movement Desensitization and Reprocessing (EMDR): protocol, empirical status, and conceptual issues". Journal of anxiety disorders. 13 (1-2): 69–85. doi:10.1016/S0887-6185(98)00040-1. PMID 10225501.  edit
  59. ↑ Manfield P (2003). EMDR Casebook (2nd ed.). New York: W.W. Norton. ISBN 9780393704167. 
  60. ↑ 60.0 60.1 Tinker, R.; Wilson S. (1999). Through the eyes of a child: EMDR with children. New York: W.W. Norton. ISBN 0393702871. 
  61. ↑ 61.0 61.1 Greenwald R (1999). Eye movement desensitization and reprocessing in child and adolescent psychotherapy. New York: Norton. ISBN 0765702177. 
  62. ↑ Scott CV; Briere J (2006). Principles of Trauma Therapy : A Guide to Symptoms, Evaluation, and Treatment. Thousand Oaks, California: Sage Publications. p. 312. ISBN 0-7619-2921-5. 
  63. ↑ Shapiro, F. (2002). "EMDR 12 years after its introduction: past and future research". Journal of clinical psychology. 58 (1): 1–22. doi:10.1002/jclp.1126. PMID 11748594.  edit
  64. ↑ Grand D (2001). Emotional Healing at Warp Speed: The Power of EMDR. New York: Harmony Books. ISBN 0609607464. 
  65. ↑ Devilly, GJ; Spence, SH (1999). "The relative efficacy and treatment distress of EMDR and a cognitive-behavior trauma treatment protocol in the amelioration of posttraumatic stress disorder". Journal of anxiety disorders. 13 (1-2): 131–57. doi:10.1016/S0887-6185(98)00044-9. PMID 10225505.  edit
  66. ↑ Devilly GJ (2002). "Eye Movement Desensitization and Reprocessing: A chronology of its development and scientific standing". Scientific Review of Mental Health Practice. 1: 113–138. 
  67. ↑ Rogers, S.; Silver, S. (2002). "Is EMDR an exposure therapy? A review of trauma protocols". Journal of clinical psychology. 58 (1): 43–59. doi:10.1002/jclp.1128. PMID 11748596.  edit
  68. ↑ Smyth NJ; Poole AD (2002). EMDR and cognitive-behavior therapy: Exploring convergence and divergence. Shapiro. pp. 151–180. 
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  70. ↑ Schubert, S.; Lee, C. W. (2009). "Adult PTSD and Its Treatment with EMDR: A Review of Controversies, Evidence, and Theoretical Knowledge". Journal of EMDR Practice and Research. 3: 117. doi:10.1891/1933-3196.3.3.117.  edit
  71. ↑ Jaycox, LH; Foa, EB; Morral, AR (1998). "Influence of emotional engagement and habituation on exposure therapy for PTSD". Journal of consulting and clinical psychology. 66 (1): 185–92. doi:10.1037/0022-006X.66.1.185. PMID 9489273.  edit
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  73. ↑ Johnstone, K.; Page, A. (2004). "Attention to phobic stimuli during exposure: the effect of distraction on anxiety reduction, self-efficacy and perceived control". Behaviour Research and Therapy. 42 (3): 249–275. doi:10.1016/S0005-7967(03)00137-2. PMID 14975769.  edit
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  75. ↑ Rodriguez, B.; Craske, M. (1995). "Does distraction interfere with fear reduction during exposure? A test among animal-fearful subjects". Behavior Therapy. 26: 337. doi:10.1016/S0005-7894(05)80109-1.  edit
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  77. ↑ Cahill, S. (1999). "Does EMDR Work? And if so, Why? A Critical Review of Controlled Outcome and Dismantling Research". Journal of Anxiety Disorders. 13: 5–1. doi:10.1016/S0887-6185(98)00039-5.  edit
  78. ↑ Shapiro, F. (1989). "Efficacy of the eye movement desensitization procedure in the treatment of traumatic memories". Journal of Traumatic Stress. 2: 199–199. doi:10.1002/jts.2490020207.  edit
  79. ↑ Gosselin, P.; Matthews, W. (1995). "Eye movement desensitization and reprocessing in the treatment of test anxiety: A study of the effects of expectancy and eye movement". Journal of Behavior Therapy and Experimental Psychiatry. 26 (4): 331–337. doi:10.1016/0005-7916(95)00038-0. PMID 8675720.  edit
  80. ↑ MacCulloch, M. (2006). "Effects of EMDR on previously abused child molesters: Theoretical reviews and preliminary findings from Ricci, Clayton, and Shapiro". Journal of Forensic Psychiatry and Psychology. 17: 531–537. doi:10.1080/14789940601075760.  edit
  81. ↑ Salkovskis, P (2002). "Review: eye movement desensitization and reprocessing is not better than exposure therapies for anxiety or trauma". Evidence-based mental health. 5 (1): 13. doi:10.1136/ebmh.5.1.13. PMID 11915816.  edit
  82. ↑ Barrowcliff, A.; Gray, N.; MacCulloch, S.; Freeman, T.; MacCulloch, M. (2003). "Horizontal rhythmical eye movements consistently diminish the arousal provoked by auditory stimuli". The British journal of clinical psychology / the British Psychological Society. 42 (Pt 3): 289–302. doi:10.1348/01446650360703393. PMID 14565894.  edit
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