Intra-aortic balloon pump

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An intra-aortic balloon pump

The Intra-aortic balloon pump (IABP) is a mechanical device that decreases myocardial oxygen demand while at the same time increasing cardiac output. Increasing cardiac output increases coronary blood flow and therefore myocardial oxygen delivery. It consists of a cylindrical polyethylene balloon that sits in the aorta, approximately 2 centimeters (0.8 in) from the left subclavian artery and counterpulsates. That is, it actively deflates in systole, increasing forward blood flow by reducing afterload. It actively inflates in diastole, increasing blood flow to the coronary arteries. These actions combine to decrease myocardial oxygen demand and increase myocardial oxygen supply.

A computer-controlled mechanism inflates the balloon with helium from a cylinder during diastole, usually linked to either an electrocardiogram (ECG) or a pressure transducer at the distal tip of the catheter; some IABPs, such as the Datascope System 98XT, allow asynchronous counterpulsation at a set rate, though this setting is rarely used. Helium is used because its low viscosity allows it to travel quickly through the long connecting tubes, and has a lower risk of causing an embolism should the balloon rupture.

History

The IABP device was pioneered at Grace Sinai Hospital in Detroit during the early 1960s by Dr. Adrian Kantrowitz and his team. The device was developed for use in heart surgery by Dr. David Bregman in 1976 at NewYork-Presbyterian Hospital in New York City.

The first clinical implant was performed at Maimonides Hospital, Brooklyn, N.Y. in Oct., 1967. The patient, a 48 year old woman, was in cardiogenic shock and unresponsive to traditional therapy. An IABP was inserted by a cut down on the left femoral artery. Pumping was performed for approximately 6 hours. Shock reversed and the patient was discharged.

The size of the original ballon was 15 French but eventually 9 and 8 French balloons were developed. A second operation removed the balloon. Since 1979 the placement of the balloon has been modified using the Seldinger technique.

Indications

The following situations may benefit from this device.

• Cardiogenic shock when used alone as treatment for myocardial infarction. 9-22% survive the first year.
• Reversible intracardial mechanical defects complicating infarction, i.e. acute mitral regurgitation and septal perforation.
• Unstable angina pectoris benefits from counterpulsation.
• Post cardiothoracic surgery—most common and useful is counterpulsation in weaning patients from cardiopulmonary bypass after continued perioperative injury to myocardial tissue.
• Preoperative use is suggested for high-risk patients such as those with unstable angina with stenosis greater than 70% of main coronary artery, in ventricular dysfunction with an ejection fraction less than 35%.
• Percutaneous coronary angioplasty
• In high risk coronary artery bypass graft surgery where cardiopulmonary bypass time was shortened, as well as during intubation period and hospital stay.
• Thrombolytic therapy of acute myocardial infarction.

Contraindications

Absolute contraindication

The following conditions will always exclude patients for treatment:

• Severe aortic valve insufficiency
• Aortic dissection
• Severe aortoiliac occlusive disease

Relative contraindication

The following conditions could, under pressing circumstances, indicate IABP therapy:

• Prosthetic vascular grafts in the aorta
• Aortic aneurysm
• Aortofemoral grafts

Complications

Since the device is placed in the femoral artery and aorta it could provoke ischemia, and compartment syndrome. The leg is at highest risk because it is supplied by the femoral artery may become ischemic. Placing the balloon too distal from the arcus aortae may induce occlusion of the renal artery and subsequent renal failure. Other possible complications are cerebral embolism during insertion, infection, dissection of the aorta or iliac artery, perforation of the artery and hemorrhage in the mediastinum. Mechanical failure of the balloon itself is also a risk which entails vascular surgery to remove under that circumstance. After balloon removal there is also a risk of 'embolic shower' from micro clots that have formed on the surface of the balloon, and can lead to peripheral thrombosis, myocardial ischemia, hemodynamic decompensation, and late pseudoaneurysm.

See also

• Cardiogenic shock
• Ventricular assist device

External links

• Intra-italheartj.org/pdf_files/20050080.pdf Images in cardiovascular medicine The missing intra-aortic balloon pump catheter by Pasquale Totaro, Nello Degno, John Smith, Vincenzo Argano, Cardiac Surgery Department, Regional Cardiac Center, Morriston Hospital, Swansea, UK, Ital Heart J 2005; 6 (4): 361-362)

de:Intraaortale Ballonpumpe