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...muscarinic [[acetylcholine receptor]]s ([[acetylcholine]] being the main [[neurotransmitter]] used by the [[parasympathetic]] nervous system). Therefore, it may cause ...upillary sphincter]] muscle, which is normally stimulated by acetylcholine release, thereby allowing the radial [[pupillary dilator]] [[muscle]] to contract a

...f needed to assess potential adverse effects on patients.<ref>{{cite press release |title=Severe Pain Relief may be Possible with a Common Anesthetic Drug |pu .... NMDA and AMPA are receptors for the [[neurotransmitter]] [[Glutamic acid#Neurotransmitter|glutamate]]. The glutamate system has been implicated in depression recentl

Valproate is believed to affect the function of the [[neurotransmitter]] [[GABA]] in the human brain, making it an alternative to [[Lithium pharma | title = FDA Issues Approvable Letter For Stavzor Delayed Release Valproic Acid Capsules

...by the [[hypothalamus]]. Its main function as a hormone is to inhibit the release of [[prolactin]] from the anterior lobe of the [[pituitary]]. ...[[norepinephrine]] (noradrenaline) and [[epinephrine]] (adrenaline), but a neurotransmitter as well.

'''Epinephrine''' (also known as '''adrenaline''') is a [[hormone]] and [[neurotransmitter]].<ref>{{cite pmid|6278965}}</ref>. It increases heart rate, contracts bloo ...aline from the cytosol back into chromaffin granules in preparation for release.{{Citation needed|date=April 2009}}

...}}</ref> The pharmacological action of diazepam enhances the effect of the neurotransmitter GABA by binding to the benzodiazepine site on the [[GABAA receptor|GABA<sub ** Capsules, time-release – 15&nbsp;mg (marketed by Roche as '''Valrelease''')<ref name="ElephantCa

...ain pharmacological action of temazepam is to increase the effect of the [[neurotransmitter]] [[GABA]] (gamma-aminobutyric acid) at the GABA_A receptor. This ...or=Welt T, Engelmann M, Renner U, ''et al.'' |title=Temazepam triggers the release of vasopressin into the rat hypothalamic paraventricular nucleus: novel ins

Natural release of the hormone [[oxytocin]] can cause mild to moderate mydriasis. Strong se ...sed for this task however. Any potent norepinephrine reuptake inhibitor or release agent should be capable of such an effect.

...{{cite journal |author=Korenke AR, Rivey MP, Allington DR |title=Sustained-release fampridine for symptomatic treatment of multiple sclerosis |journal=Ann Pha ..., prolonging [[action potentials]] and thereby increasing neurotransmitter release at the [[neuromuscular junction]].<ref>{{cite journal |author=Judge S, Beve

...he salivary glands of the [[octopus]] and has since been found to act as [[neurotransmitter]], [[neurohormone]] and [[neuromodulator]] in [[invertebrate]]s. It is wide ...ne has a major role in learning and memory. In the [[firefly]], octopamine release leads to light production in the lantern.

...ut bullets lodged in the joints are the exception, as they deteriorate and release lead into the body over time.<ref>{{cite pmid|2926538}}</ref> ...ure takes place over years, clearance is much slower, partly due to the re-release of lead from bone.<ref name="Hu07"/> Many other tissues store lead, but tho

...h, in turn are derived from [[neuroectoderm]]. They release a variety of [[neurotransmitter]]s, including [[acetylcholine]], [[adenosine triphosphate|ATP]], and [[dopa ...2+</sup> entry, excitation of glomus cells and consequent neurotransmitter release <ref name="pmid10811732>{{cite journal |author=Buckler KJ, Williams BA, Hon

...]], the parasympathetic nervous system stimulates neurons that release the neurotransmitter [[acetylcholine]] (ACh) at synapses with [[cardiomyocyte|cardiac muscle cel

...e]]s (e.g. [[Antidiuretic hormone|vasopressin]] and [[angiotensin]]) and [[neurotransmitter]]s (e.g. [[epinephrine]]) from the nervous system. Permeability of the [[endothelium]] is pivotal in the release of nutrients to the tissue. It is also increased in [[inflammation]] in res

...rocannabinol (Δ⁸-THC), mimic the action of [[anandamide]], a [[neurotransmitter]] produced naturally in the body. The THCs produce the ''high'' associated ...cellular signaling role they are similar to the well-known [[monoamine]] [[neurotransmitter]]s, such as [[acetylcholine]] and [[dopamine]], endocannabinoids differ in

...tin was initially synthesized to mimic the [[chemical structure]] of the [[neurotransmitter]] gamma-aminobutyric acid ('''GABA'''), but is not believed to act on the s {{News release section|date=March 2010}}

...at: [http://books.google.com/books?id=nNH3p29wjK4C&pg=PA116&dq=endorphins+neurotransmitter&hl=en&ei=IwwlTKfzHMr-_Aa-w6zGBA&sa=X&oi=book_result&ct=result&resnum=10&ved ...By hijacking this process, exogenous opioids cause inappropriate dopamine release, and lead to aberrant [[synaptic plasticity]], which causes addiction. Opio

...synthesize and degrade the endocannabinoids anandamide and 2-AG. Unlike [[neurotransmitter]]s, endogenous cannabinoids are not stored in [[vesicle]]s after synthesis, ...ignaling in a variety of physiological processes, including neuromodulator release,<ref>{{cite journal |author=Fortin DA, Levine ES |title=Differential effect

|title=Changes in neurotransmitter release at a neuromuscular junction of the lobster caused by cannabinoids

...lving the μ₁ [[opioid receptor]], precipitating a [[dopamine]] release in the [[nucleus accumbens]]. The effects of the drug can be suppressed by ...sion is thought to occur as follows: First, transmission of the excitatory neurotransmitter [[glutamate]] causes an influx of [[calcium]] ions into the [[post-synaptic

...properly. Drugs such as morphine change properties such as cell adhesion, neurotransmitter volume, and synaptic traffic. After significant morphine application, tyro ...o physically injuring the brain tissue, traumatic brain injury induces the release of glutamate that interacts with ionotropic glutamate receptors (iGluRs).

...onfirmation of chemical [[synapse|synaptic transmission]] and the first [[neurotransmitter]] ever discovered. It was later confirmed to be [[acetylcholine]], which wa ...later confirmed to be [[acetylcholine]] and was found to be the principal neurotransmitter in the [[parasympathetic nervous system]].

...on]]. When the wave reaches a synapse, it provokes release of a puff of [[neurotransmitter]] molecules, which bind to chemical receptor molecules located in the membr ...Eccles himself in later publications, have taken it to mean that ''neurons release the same set of transmitters at all of their synapses''.

...and voltage dependent Ca²⁺ channels (VDCC) with high glutamate release, which is taken up again by EAAT1 and EAAT2. This results in a small rise i ...s. This occurs when [[cell surface receptor|receptors]] for the excitatory neurotransmitter [[glutamic acid|glutamate]] ([[glutamate receptor]]s) such as the [[NMDA re

...neurotransmitters into vesicles. The result is increased neurotransmitter release that is not dependent on the phasic activity of the presynaptic cell. *{{cite journal |author=Kilbourn MR |title=In vivo radiotracers for vesicular neurotransmitter transporters |journal=Nucl. Med. Biol. |volume=24 |issue=7 |pages=615–9 |

...secrete with glutamate or GABA. Glutamate is excitatory, meaning that the release of glutamate by one cell usually causes adjacent cells to fire an [[action **[[Gamma-aminobutyric acid|GABA]] is an example of an inhibitory neurotransmitter.

...l-triphosphate]] (IP₃), [[IP3|IP₃]], controlling the release of intracellular calcium stores into the cytoplasm (see [[signal transducti ...; Many receptors make up this family, including [[adrenergic receptor]]s, neurotransmitter receptors, [[olfactory receptor]]s, [[opioid receptor]]s, [[chemokine recep

...antagonist]] against [[glutamate]]. Glutamate is the primary excitatory [[neurotransmitter]] in the [[central nervous system|CNS]]. NMDA receptors have a very importa ...et al. 2005. Driving capacity of patients treated with methadone and slow-release oral morphine. Addiction 100 (7): 1027.</ref> Drug abuse patients had signi

...nducted in the early 1960s by Dr. [[Henri Laborit]] to use in studying the neurotransmitter [[GABA receptor|GABA]].<ref>{{cite journal | author = H. Laborit, J.M. Joua ...ybutyric acid (GHB) and gamma-aminobutyric acidB (GABAB) receptor-mediated release of GABA and glutamate (GLU) in rat thalamic ventrobasal nucleus (VB): a pos