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  • ===Suppression of prostaglandins and thromboxanes=== Aspirin's ability to suppress the production of prostaglandins and thromboxanes is due to its irreversible inactivation of the [[cyclooxyg
    78 KB (10,918 words) - 16:52, 27 September 2010
  • 29 KB (3,955 words) - 22:17, 21 September 2010
  • {{Prostaglandins}} [[Category:Prostaglandins]]
    4 KB (610 words) - 09:43, 20 September 2010
  • ...nting an excessive rise in GFR. Angiotensin II causes the local release of prostaglandins, which, in turn, antagonize renal vasoconstriction. The net effect of these
    14 KB (1,818 words) - 21:27, 21 September 2010
  • 1 KB (126 words) - 21:28, 21 September 2010
  • 3 KB (394 words) - 21:28, 21 September 2010
  • ...cytokines]], [[anaphylatoxins]], or other mediators (such as leukotrienes, prostaglandins, histamine, bradykinin, etc.) highly influenced by the [[immune system]].
    10 KB (1,408 words) - 21:29, 21 September 2010
  • 8 KB (1,132 words) - 21:29, 21 September 2010
  • ...402-3. PMID 3032669</ref><ref>Burstein S, Hunter SA, Latham V, Renzulli L. Prostaglandins and cannabis--XVI. Antagonism of delta 1-tetrahydrocannabinol action by its
    6 KB (823 words) - 22:09, 21 September 2010
  • ...[[Enzyme inhibitor|inhibiting]] the [[Biosynthesis|bodily synthesis]] of [[prostaglandins]]. Ketorolac in its oral ([[tablet]] or [[Capsule (pharmacy)|capsule]]) an ...orolac should be avoided in patients with renal ([[kidney]]) dysfunction. (Prostaglandins are needed to dilate the [[afferent arteriole]]; NSAIDs effectively reverse
    16 KB (2,012 words) - 22:15, 21 September 2010
  • The inhibition of prostaglandins also has the effect of decreasing the protection given to the stomach from
    7 KB (976 words) - 22:15, 21 September 2010
  • ...verse renal [kidney] effects caused by the reduction in synthesis of renal prostaglandins"<!--
    28 KB (3,886 words) - 22:16, 21 September 2010
  • 3 KB (387 words) - 22:16, 21 September 2010
  • 1 KB (130 words) - 22:16, 21 September 2010
  • 11 KB (1,540 words) - 22:16, 21 September 2010
  • ...in|prostaglandins]] (cyclooxygenase), resulting in lower concentrations of prostaglandins. As a consequence, inflammation, pain and fever are reduced. The FDA approv
    8 KB (1,051 words) - 22:16, 21 September 2010
  • ...classes which could loosely be regarded as "good prostaglandins" and "bad prostaglandins", according to the structure of a particular [[enzyme]] involved in their [ ...le for maintenance and protection of the [[gastrointestinal tract]], while prostaglandins whose synthesis involves the cyclooxygenase-II enzyme, or COX-2, are respon
    30 KB (4,194 words) - 22:17, 21 September 2010
  • 3 KB (472 words) - 22:17, 21 September 2010
  • ...n. By blocking cyclo-oxygenase, dexketoprofen prevents the production of [[prostaglandins]] and therefore reduces inflammation and pain. Along with Peripheral analge
    5 KB (653 words) - 22:17, 21 September 2010
  • ...rom the cellular [[phospholipid]] bilayer by phospholipase A<sub>2</sub>). Prostaglandins act (among other things) as messenger molecules in the process of [[inflamm ...s the precursor substrate for cyclooxygenase leading to the production of prostaglandins F,D & E.
    35 KB (4,649 words) - 22:18, 21 September 2010